Epithelial ovarian cancer, fallopian tube cancer, primary peritoneal cancer

Active Ingredient: Rucaparib

Indication for Rucaparib

Population group: only adults (18 years old or older)
Therapeutic intent: Curative procedure

Rucaparib is indicated as monotherapy for the maintenance treatment of adult patients with advanced (FIGO Stages III and IV) high-grade epithelial ovarian, fallopian tube, or primary peritoneal cancer who are in response (complete or partial) following completion of first-line platinum-based chemotherapy.

Rucaparib is indicated as monotherapy for the maintenance treatment of adult patients with platinum-sensitive relapsed high-grade epithelial ovarian, fallopian tube, or primary peritoneal cancer who are in response (complete or partial) to platinum-based chemotherapy.

For this indication, competent medicine agencies globally authorize below treatments:

600 mg twice daily

For:

Dosage regimens

Oral, 600 milligrams rucaparib, 2 times daily.

Detailed description

The recommended dose of rucaparib is 600 mg taken twice daily, equivalent to a total daily dose of 1 200 mg.

Patients should start the maintenance treatment with rucaparib no later than 8 weeks after completion of their final dose of the platinum containing regimen.

Duration of treatment

First-line maintenance treatment of advanced ovarian cancer

Patients can continue treatment until disease progression, unacceptable toxicity or completion of 2 years treatment.

Maintenance treatment of platinum-sensitive relapsed ovarian cancer

Patients can continue treatment until disease progression or unacceptable toxicity.

If a patient vomits after taking rucaparib, the patient should not retake the dose and should take the next scheduled dose.

Missed doses

If a dose is missed, the patient should resume taking rucaparib with the next scheduled dose.

Dose adjustments for adverse reactions

Adverse reactions may be managed through dose interruptions and/or dose reductions for moderate to severe reactions (i.e. CTCAE Grade 3 or 4) such as neutropenia, anaemia and thrombocytopenia.

Liver transaminase elevations (aspartate aminotransferase (AST) and/or alanine aminotransferase (ALT)) occur early in treatment and are generally transient. Grade 1 to 3 elevations in AST/ALT can be managed without change to the rucaparib dose, or with treatment modification (interruption and/or dose reduction). Grade 4 reactions require treatment modification (see Table 2).

Other moderate to severe non-haematological adverse reactions such as nausea and vomiting, can be managed through dose interruption and/or reductions, if not adequately controlled by appropriate symptomatic management.

Table 1. Recommended dose adjustments:

Dose reductionDose
Starting dose600 mg twice daily (two 300 mg tablets twice daily)
First dose reduction500 mg twice daily (two 250 mg tablets twice daily)
Second dose reduction400 mg twice daily (two 200 mg tablets twice daily)
Third dose reduction300 mg twice daily (one 300 mg tablet twice daily)

Table 2. Management of Treatment-emergent AST/ALT Elevations:

Grade of AST/ALT ElevationManagement
Grade 3 without other signs of liver dysfunctionMonitor LFTs weekly until resolution to
Grade ≤ 2
Continue rucaparib provided bilirubin is < ULN
and alkaline phosphatase is < 3 × ULN
Interrupt treatment if AST/ALT levels do not
decline within 2 weeks until Grade ≤ 2, then
resume rucaparib at the same or at a reduced
dose
Grade 4Interrupt rucaparib until values return to
Grade ≤ 2; then resume rucaparib with a dose
reduction and monitor LFTs weekly for 3 weeks

Elderly

No adjustment is recommended to the starting dose for elderly patients (≥65 years of age). Greater sensitivity of some elderly patients (≥65 years of age) to adverse events cannot be ruled out. There are limited clinical data in patients aged 75 or over.

Dosage considerations

The doses should be taken approximately 12 hours apart.

Active ingredient

Rucaparib

Rucaparib is an inhibitor of poly(ADP-ribose) polymerase (PARP) enzymes, including PARP-1, PARP-2, and PARP-3, which play a role in DNA repair. In vitro studies have shown that rucaparibinduced cytotoxicity involves inhibition of PARP enzymatic activity and the trapping of PARP-DNA complexes resulting in increased DNA damage, apoptosis, and cell death.

Read more about Rucaparib

Related medicines

Develop a tailored medication plan for your case, considering factors such as age, gender, and health history

Ask the Reasoner

Liability Disclaimer : RxReasoner has utilized reasonable care in providing content and services that are accurate, complete and up to date. However, RxReasoner does not accept any responsibility or liability about it. The content and services of RxReasoner are for informational purposes only and they are not intended to be a substitute for the knowledge, expertise, skill, and judgment of physicians, pharmacists, nurses, or other healthcare professionals involved in patient care. RxReasoner offers no medical advice. Users are responsible for the use of the provided content. A shown indication or treatment should not be construed to indicate that the medication is safe, appropriate, or effective in any given patient or under any particular circumstances. The absence of an indication or treatment should not roule out the existence of other appropriate medications. Always seek the advice of a physician or other qualified health provider with any questions you may have regarding a medical condition or medicament. RxReasoner is not liable for any damages allegedly sustained arising out of the use of its content and services.

⇈